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    Overexpression of microRNA-30a inhibits hepatitis B virus X protein-induced autophagosome formation in hepatic cells

    New search for: Kumar, S.
    New search for: Gupta, P.
    New search for: Khanal, S.
    New search for: Shahi, A.
    New search for: Kumar, P.
    New search for: Sarin, S. K.
    New search for: Venugopal, S. K.

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    Overexpression of microRNA-30a inhibits hepatitis B virus X protein-induced autophagosome formation in hepatic cells

    New search for: Kumar, S.
    New search for: Gupta, P.
    New search for: Khanal, S.
    New search for: Shahi, A.
    New search for: Kumar, P.
    New search for: Sarin, S. K.
    New search for: Venugopal, S. K.

    Access

    Access via TIB
    Check availability in my library
    Order at Subito €

    Overexpression of microRNA-30a inhibits hepatitis B virus X protein-induced autophagosome formation in hepatic cells

    New search for: Kumar, S.
    New search for: Gupta, P.
    New search for: Khanal, S.
    New search for: Shahi, A.
    New search for: Kumar, P.
    New search for: Sarin, S. K.
    New search for: Venugopal, S. K.

    Access

    Access via TIB
    Check availability in my library
    Order at Subito €

    Access

    Access via TIB
    Check availability in my library
    Order at Subito €

    Page navigation

    Document information
    Similar titles

    Export, share and cite

    Document information
    Title:

    Overexpression of microRNA-30a inhibits hepatitis B virus X protein-induced autophagosome formation in hepatic cells

    Contributors:

    Kumar, S. (author) / Gupta, P. (author) / Khanal, S. (author) / Shahi, A. (author) / Kumar, P. (author) / Sarin, S. K. (author) / Venugopal, S. K. (author)

    Published in:

    FEBS JOURNAL ; 282 ; 1152-1163

    Publication date:

    2015-01-01

    Size:

    12 pages

    ISSN:

    1742-464X

    Type of media:

    Article (Journal)

    Type of material:

    Print

    Language:

    English

    Classification:

    DDC:  572

    © Metadata Copyright the British Library Board and other contributors. All rights reserved.

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